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Cytochrome C is used in first aid or adjuvant therapy for tissue hypoxia for a variety of reasons. It also has a certain therapeutic effect on the reduction of white blood cells caused by anti-cancer drugs, limb circulation disorders, liver diseases, and nephritis.
| Items | Specification | Result |
| Appearance | Red or red-brown crystalline powder | Red crystalline powder |
| PH | 6.0-7.5 | 6.16 |
| Assay | ≥90.0% | 91.6% |
| Loss on drying | ≤5.0% | 3.51% |
| Residue on ignition | ≤2.0% | 1.05% |
| Product parameters | |
| Cas number: | 9007-43-6 |
| Appearance: | Red or red-brown crystalline powder |
| Purity: | 90%min |
| MOQ: | 1g |
| Price: | Negotiable |
| Package details: | 1g/bag or bottle |
| Brand: | Fortunachem |
Cytochrome c is a small, water-soluble heme protein essential for life, found in the mitochondria of nearly all eukaryotes and some bacteria. Here's a concise overview:
Structure: Contains a heme group (iron protoporphyrin IX) where iron alternates between Fe²⁺ (reduced) and Fe³⁺ (oxidized). Its protein structure is highly conserved evolutionarily.
Location: Located in the intermembrane space of mitochondria, loosely associated with the inner membrane.
Primary Function (Energy Production):
Key component of the mitochondrial electron transport chain (Respiratory Chain).
Shuttles electrons from Complex III (cytochrome bc₁ complex) to Complex IV (cytochrome c oxidase).
Crucial for aerobic cellular respiration, driving ATP synthesis via oxidative phosphorylation.
Critical Secondary Function (Cell Death):
Plays a central role in intrinsic apoptosis (programmed cell death).
Released from mitochondria into the cytosol in response to cell stress/damage signals.
Binds Apaf-1 in the cytosol, triggering formation of the apoptosome, which activates caspases leading to controlled cell death.
Significance:
Evolution: Extreme amino acid sequence conservation makes it a "molecular clock" for studying evolution.
Medicine: Used as a biomarker for cell damage (e.g., elevated in serum after heart attacks). Research explores its therapeutic potential in ischemia-reperfusion injury and roles in diseases involving mitochondrial dysfunction or dysregulated apoptosis (e.g., cancer, neurodegeneration).
In essence, cytochrome c is a vital electron carrier for energy production and a crucial signaling molecule for initiating programmed cell death.
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